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Experimental data indicates that the peptide fragment interacts with the 3-adrenergic pathway, which mediates the breakdown of triglycerides into free fatty acids and glycerol within adipose tissue

Via drainage into the portal circulation, VAT floods the liver with FFAs and inflammatory mediators, exacerbating hepatic IR, promoting dyslipidemia, and stimulating the production of acute-phase proteins like CRP, thereby amplifying the systemic inflammatory burden (38, 54)

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ISCs are involved in electron transport, substrate binding, enzymatic catalysis and play critical roles in maintaining genomic stability since several DNA polymerases and helicases require interaction with ISCs for proper functioning