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(2017) found that the expression of ASPH was upregulated in tamoxifen-resistant cells, and the upregulation depended on the PI3K and MAPK pathways
Two IGF-I analogs that bind poorly to IGF-binding proteins, the truncated form, des(1-3)IGF-I, and a variant with an N-terminal extension as well as arginine at residue 3, LR3IGF-I, were approx
Crosstalk of signaling and metabolism mediated by the NAD + /NADH redox state in brain cells
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doi: 10.4049/jimmunol.1800085 238 YangDYangLCaiJHuXLiHZhangXet al
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